|Planning to fry your legs? Ingest 0.3g/kg sodium bicarbonate before your workout!|
In a recent study, Jason C. Siegler nd Paul Marshall from the School of Science and Health at the University of Western Sydney determined whether inducing metabolic alkalosis differentially affects descending central drive after fatiguing exercise and whether this effect may, in part, be explained by attenuating group III and IV afferent firing.
In the corresponding experiment eleven recreationally active, young, male subjects performed a maximal 2 min voluntary knee extension (MVC) followed by a 2 min rest and subsequent 1 min MVC with an occlusive cuff either in placebo [PLA; 0.3 g/kg body weight calcium carbonate] or alkalosis conditions [ALK; 0.3 g/kg body weight sodium bicarbonate].
"At each of the two experimental sessions, a pre-exercise standardized beverage consisting of 0.3 g/kg BW of 60% maltodextrin and 40% protein (casein and whey) was consumed 1 h before commencing the ingestion protocol [...] After 1 h quiet rest, participants consumed the first of three equally divided doses of either PLA- or ALK-filled gelatin capsules. Capsules were consumed at −90, −60 and −30 min before the exercise protocol" (Siegler. 2015).For those of you who are having trouble with bicarbonate and diarrhea or general gastro-intestinal distress it may be interesting to know that Siegler et al. have found in previous studies that the preload with the CHO + PRO beverage minimizes the potential for subjects to experience the negative side-effects often associated with ingesting NaHCO3 (Siegler. 2010).
|Base excess after ingestion of different amounts of NaHCO3 = sodium bicarbonate (Siegler. 2010)|
Ah, there's something else I wan to address: Let's also take a look at the stupid non-alternatives out there. Beta-alanine is no alkalizer. It works (if at all) in the cell and may have additive effects, but cannot replace sodium bicarbonate. What is even more stupid, though, is to suggest using calcium carbonate instead. Don't laugh, I've seen "experts" suggest that, because "it's healthier than sodium!" First of all, it's not healthier. Second of all, it's not working AT ALL. In fact it is used as placebo in sodium bicarbonate studies like the one at hand. Ah, and speaking of stupid non-functional replacements. If you want to decrease your performance, go ahead and use potassium carbonate instead. The exuberant increase in serum potassium from supplementation and exercise which will increase the eflux of potassium from the cell into the blood (Medbø. 1990) may eventually impair not increase your performance (Lindinger. 1995). So goddamn, forget about the myth of the bad sodium and stick to what works: Sodium bicarbonate, the only form of sodium that has proven hypotensive (=blood pressure lowering) effects (Luft. 1990).
"The actual exercise protocol was performed on an isokinetic dynamometer (KinCom 125, version 5.32, Chattanooga, TN, USA). Participants were seated with their hip and leg flexed to 90 deg, the centre of rotation of the lever arm was carefully aligned with the sagittal plane axis of the knee joint, the lever arm of the dynamometer was firmly attached 2–3 cm superior to the lateral malleolus, and the participant was firmly strapped into the chair with straps across the trunk. [...] Before the exercise protocol, participants performed a series of submaximal isometric knee-extension efforts (25, 50 and 75% of perceived maximal effort). Six MVCs [maximal voluntare contractions] were then performed in a random order (two with stimulation applied, two with cuff and stimulation applied, and two with no additional measures performed), with 2 min rest between efforts. Each MVC was required to be as fast and forceful as possible and maintained for 3–4 s" (Siegler. 2015).The results of the extensive tests Siegler and Marshall conducted were unambiguous: While the voluntary activation declined to a similar extent after the 2 min MVC, the voluntary activation during the ischaemic period, i.e. during a period of reduced oxygen availability as it may occur towards the end of a long, intense workout, the voluntary activation was higher during the sodium bicarbonate aka ALK (PLA, 57 ± 8%; ALK, 76 ± 5%) trial.
- Luft, Friedrich C., et al. "Sodium bicarbonate and sodium chloride: effects on blood pressure and electrolyte homeostasis in normal and hypertensive man." Journal of hypertension 8.7 (1990): 663-670.
- Medbø, J. I., and O. M. Sejersted. "Plasma potassium changes with high intensity exercise." The Journal of Physiology 421.1 (1990): 105-122.
- Lindinger, Michael I. "Potassium regulation during exercise and recovery in humans: implications for skeletal and cardiac muscle." Journal of molecular and cellular cardiology 27.4 (1995): 1011-1022.
- Siegler, Jason C., et al. "Effects of various sodium bicarbonate loading protocols on the time-dependent extracellular buffering profile." The Journal of Strength & Conditioning Research 24.9 (2010): 2551-2557.
- Siegler, Jason C., and Paul Marshall. "The effect of metabolic alkalosis on central and peripheral mechanisms associated with exercise induced muscle fatigue in humans." Experimental physiology (2015).